[1] Rang Z,Wang ZY,Pang QY,et al.MiR-181a targets PHLPP2 to augment AKT signaling and regulate proliferation and apoptosis in human keloid fibrobla.Sts[J].Cell Physiol Biochem,2016,40(3-4):796~806. [2] Luan Y, Liu Y, Liu C, et a1.Serum miRNAs signature plays an important role in keloid disease[J].Curr Mol Med,2016,16(5):504~514. [3] Yu X1, Li Z2, Chan MT3, et a1.microRNA deregulation in keloids: an opportunity for clinical intervention[J].Cell Prolif,2015,48(6):626~630. [4] Banmjohann D,Ansel KM.MicroRNA regulation of the germinal center response[J].Curt OpinImmunol,2014,6(28):6~11. [5] 蔡景龙.瘢痕疙瘩发生的肿瘤源性学说[J].中华医学杂志,2009,89(16):1084~1087. [6] Vincent AS1, Phan TT, Mukhopadhyay A, et a1.Human skin keloid fibroblasts display bioenergetics of cancercells[J].Invest Dermatol,2008,128(3):702~709. [7] 宁璞,刘德伍,毛远桂等.增生性瘢痕与正常皮肤微小RNA的差异表达谱分析[J].中华医学杂志,2012,92(10):692~694. [8] Li C,Bai Y,Liu H,et a1.Comparative study of microRNA profiling in keloid fibroblast and annotation of differential expressedmicroRNAs[J].Acta Biochim Biophys Sin,2013,45(8):692~699. [9] SachdevaM,Mo YY. miR-145-mediated suppression of cellgrowth,invasion andmetastasis[J]. Am Transl Res,2010,2(2):170~180. [10] Fang S, Xu C, Zhang Y,et a1.Umbilical cord-derived mesenchymal stem cell-derived exosomal microRNAs suppress myofibroblast differentiation by inhibiting the transforming growth factor-β/SMAD2 pathway during wound healing[J].Stem Cells Transl Med,2016,5(10):1425~1439. [11] Gras C, Ratuszny D, Hadamitzky C,et a1.miR-145 contributes to hypertrophic scarring of the skin by inducing myofibroblast activity[J].Mol Med,2015, 9(21):296~304. [12] 肖刚,王智园,谭敏,等.转化生长因子β1对病理性瘢痕中成纤维细胞增殖及凋亡水平的影响[J].实用医学杂志,2008,24(13):2242~2245. [13] 武继祥,陈德英,吴宗耀.增生性瘢痕中TGF-β和胶原酶、TIMP-1 mRNA表达的定量研究[J].中华整形外科杂志,2000,16(1):34. [14] 徐孝兴.病理性瘢痕与TGF-β介导的信号通路的相关性研究进展[J].齐齐哈尔医学院学报,2013,34(1):77~80. [15] Babalola O, Mamalis A, Lev-Tov H,et a1. The Role of microRNAs in skin fibrosis[J].Arch Dermatol Res. 201,305(9):763~776.
2019, Vol. 25 
冀公网安备13080202000677号