[1] Jan N,Dana R,Kazuo T,et al.New insights into the pathogenesis of IgA nephropathy[J].Kidney Diseases,2015,1(1):8~18. [2] Magistroni R, D'Agati VD, Appel GB, et al.New developments in the genetics, pathogenesis, and therapy of IgA nephropathy[J].Kidney Int,2015,88:974~989. [3] Yeo SC, Cheung CK, Barratt J.New insights into the pathogenesis of IgA nephropathy[J].Pediatr Nephrol,2018,33(5):763~777. [4] Knoppova B, Reily C, Maillard N, et al.The origin and activities of IgA1-containing immune complexes in IgA nephropathy[J].Front Immunol,2016,7:117. [5] Sun Q, Zhang J, Zhou N, et al.DNA methylation in cosmc promoter region and aberrantly glycosylated IgA1 associated with pediatric IgA nephropathy[J].PLoS One,2015,10:e0112305. [6] Sun Y, Liu Z, Liu Y, et al.Increased frequencies of memory and activated B cells and follicular helper T cells are positively associated with high levels of activation-induced cytidine deaminase in patients with immunoglobulin A nephropathy[J].Mol Med Rep,2015,12:5531~5537. [7] Meng H, Zhang L, Ye EX, et al.Application of Oxford classification, and overexpression of transforming growth factor-β1 and immunoglobulins in immunoglobulin A nephropathy:correlation with World Health Organization classification of immunoglobulin A nephropathy in a Chinese patient cohort[J].Transl Res,2014,163:8~18. [8] Zhai Y-L, Zhu L, Shi S-F, et al.Increased APRIL expression induces IgA1 aberrant glycosylation in IgA nephropathy[J].Medicine (Baltimore),2016,95:e3099. [9] Liu Y, Liu H, Peng Y, et al.New insights into the pathogenesis of IgA nephropathy:do toll like receptor 9-B cell activation factor-IgA class switching recombination signaling axis induce IgA hyper-production[J].Ren Fail,2014,36:970~973. [10] Mestecky J, Novak J, Moldoveanu Z, et al.IgA nephropathy enigma[J].Clin Immunol,2016,172:72~77. [11] Faria B, Henriques C, Matos AC, et al.Combined C4d and CD3 immunostaining predicts immunoglobulin (Ig)A nephropathy progression[J].Clin Exp Immunol,2015,179:354~361. [12] Xiao C, Zhou Q, Li X, et al.Differentiation and recruitment of IL-22-producing helper T cells in lgA nephropathy[J].Am Transl Res,2016,8:3872~3882. [13] Gan L, Zhou Q, Li X, et al.Intrinsic renal cells induce lymphocytosis of Th22 cells from IgA nephropathy patients through B7-CTLA-4 and CCL-CCR pathways[J].Mol Cell Biochem,2018,441:191~199. [14] Jin L-W, Ye H-Y, Xu X-Y, et al.MiR-133a/133b inhibits Treg differentiation in IgA nephropathy through targeting FOXP3[J].Biomed Pharmacother,2018,101:195~200. [15] Huang H, Sun W, Liang Y, et al.CD4(+)CD25(+)Treg cells and IgA nephropathy patients with tonsillectomy:a clinical and pathological study[J].Int Urol Nephrol,2014,46:2361~2369. [16] Jacob M, Ohl K, Goodarzi T, et al.CTLA-4 polymorphisms in patients with IgA nephropathy correlate with proteinuria[J].Kidney Blood Press Res,2018,43:360~366. |